2024

Sartania, Nana ORCID: https://orcid.org/0000-0002-3196-2312, Sneddon, Sharon ORCID: https://orcid.org/0000-0001-9767-4180 and Courtney, Natalie (2024) Study techniques - a useful insight. Surgo, 2(1), (doi: 10.36399/Surgo.2.319)

2021

Courtney, Natalie, Welsh, Michelle and Varsou, Ourania ORCID: https://orcid.org/0000-0002-7789-1185 (2021) Hybrid co-teaching using technology: could this be a model for peer-mentoring? In: Tasler, Nathalie, O'Brien, Rachelle and Spiers, Alex (eds.) Being creative in the face of adversity - Annual #creativeHE collection 2021. Creativity for Learning in Higher Education community (#creativeHE), pp. 65-68. (doi: 10.25416/NTR.17709860.v4)

2019

Deguise, M.-O. et al. (2019) Abnormal fatty acid metabolism is a core component of spinal muscular atrophy. Annals of Clinical and Translational Neurology, 6(8), pp. 1519-1532. (doi: 10.1002/acn3.50855) (PMID:31402618) (PMCID:PMC6689695)

Courtney, Natalie L., Mole, Alannah J., Thomson, Alison K. and Murray, Lyndsay M. (2019) Reduced P53 levels ameliorate neuromuscular junction loss without affecting motor neuron pathology in a mouse model of spinal muscular atrophy. Cell Death and Disease, 10(7), 515. (doi: 10.1038/s41419-019-1727-6) (PMID:31273192) (PMCID:PMC6609617)

2018

Groen, Ewout J.N., Perenthaler, Elena, Courtney, Natalie L., Jordan, Crispin Y., Shorrock, Hannah K., van der Hoorn, Dinja, Huang, Yu-Ting, Murray, Lyndsay M., Viero, Gabriella and Gillingwater, Thomas H. (2018) Temporal and tissue-specific variability of SMN protein levels in mouse models of spinal muscular atrophy. Human Molecular Genetics, 27(16), pp. 2851-2862. (doi: 10.1093/hmg/ddy195) (PMID:29790918) (PMCID:PMC6077828)

2015

Murray, Lyndsay M., Beauvais, Ariane, Gibeault, Sabrina, Courtney, Natalie L. and Kothary, Rashmi (2015) Transcriptional profiling of differentially vulnerable motor neurons at pre-symptomatic stage in the Smn (2b/-) mouse model of spinal muscular atrophy. Acta Neuropathologica Communications, 3, 55. (doi: 10.1186/s40478-015-0231-1) (PMID:26374403) (PMCID:PMC4570693)

Articles

Sartania, Nana ORCID: https://orcid.org/0000-0002-3196-2312, Sneddon, Sharon ORCID: https://orcid.org/0000-0001-9767-4180 and Courtney, Natalie (2024) Study techniques - a useful insight. Surgo, 2(1), (doi: 10.36399/Surgo.2.319)

Deguise, M.-O. et al. (2019) Abnormal fatty acid metabolism is a core component of spinal muscular atrophy. Annals of Clinical and Translational Neurology, 6(8), pp. 1519-1532. (doi: 10.1002/acn3.50855) (PMID:31402618) (PMCID:PMC6689695)

Courtney, Natalie L., Mole, Alannah J., Thomson, Alison K. and Murray, Lyndsay M. (2019) Reduced P53 levels ameliorate neuromuscular junction loss without affecting motor neuron pathology in a mouse model of spinal muscular atrophy. Cell Death and Disease, 10(7), 515. (doi: 10.1038/s41419-019-1727-6) (PMID:31273192) (PMCID:PMC6609617)

Groen, Ewout J.N., Perenthaler, Elena, Courtney, Natalie L., Jordan, Crispin Y., Shorrock, Hannah K., van der Hoorn, Dinja, Huang, Yu-Ting, Murray, Lyndsay M., Viero, Gabriella and Gillingwater, Thomas H. (2018) Temporal and tissue-specific variability of SMN protein levels in mouse models of spinal muscular atrophy. Human Molecular Genetics, 27(16), pp. 2851-2862. (doi: 10.1093/hmg/ddy195) (PMID:29790918) (PMCID:PMC6077828)

Murray, Lyndsay M., Beauvais, Ariane, Gibeault, Sabrina, Courtney, Natalie L. and Kothary, Rashmi (2015) Transcriptional profiling of differentially vulnerable motor neurons at pre-symptomatic stage in the Smn (2b/-) mouse model of spinal muscular atrophy. Acta Neuropathologica Communications, 3, 55. (doi: 10.1186/s40478-015-0231-1) (PMID:26374403) (PMCID:PMC4570693)

Book Sections

Courtney, Natalie, Welsh, Michelle and Varsou, Ourania ORCID: https://orcid.org/0000-0002-7789-1185 (2021) Hybrid co-teaching using technology: could this be a model for peer-mentoring? In: Tasler, Nathalie, O'Brien, Rachelle and Spiers, Alex (eds.) Being creative in the face of adversity - Annual #creativeHE collection 2021. Creativity for Learning in Higher Education community (#creativeHE), pp. 65-68. (doi: 10.25416/NTR.17709860.v4)